{"id":40731,"date":"2026-02-19T15:29:05","date_gmt":"2026-02-19T15:29:05","guid":{"rendered":"https:\/\/solongevity.com\/longevity-news\/is-longevity-written-in-our-dna-here-is-how-much-genetics-really-matters-in-the-length-of-our-lives\/"},"modified":"2026-10-16T16:10:05","modified_gmt":"2026-10-16T16:10:05","slug":"longevity-genetics-dna","status":"publish","type":"post","link":"https:\/\/solongevity.com\/en\/longevity-news\/longevity-genetics-dna\/","title":{"rendered":"Is Longevity Written in Our DNA? Here Is How Much Genetics Really Matters in the Length of Our Lives"},"content":{"rendered":"<p>How much does <b>genetics <\/b>weigh on <b>longevity<\/b>? In other words: is it our <b>genes <\/b>that determine how long we will live? For decades, the scientific community has debated this question, with studies attributing to <b>DNA <\/b>a modest influence, estimated <b>at between 6% and 25%<\/b>. Now, however, a rigorous new study conducted by an international group of scientists and published in <a href=\"https:\/\/www.science.org\/doi\/10.1126\/science.adz1187\" target=\"_blank\" rel=\"noopener\"><i>Science <\/i><\/a>suggests that these estimates are too low. Once the data are corrected, it emerges that the <b>heritability of intrinsic human lifespan would be about 50-55%<\/b>, a value in line with other complex physiological traits.    <\/p>\n<h2><b>Heritability and mortality from accidental causes<\/b><\/h2>\n<p>To understand this discovery, we need to distinguish between two types of mortality:<\/p>\n<ul>\n<li aria-level=\"1\"><b>Extrinsic Mortality:<\/b> Deaths caused by factors external to the body, such as accidents, homicides or infectious diseases.<\/li>\n<li aria-level=\"1\"><b>Intrinsic Mortality:<\/b> Deaths resulting from internal biological processes, such as cellular decline and age-related degenerative diseases.<\/li>\n<\/ul>\n<p><b>Heritability<\/b> is a statistical measure that indicates how much of the variation in a trait (in this case, <b>longevity<\/b>) in a given population is due to genetic differences. The problem with previous studies, the authors write, is that they were often based on historical cohorts (18th and 19th centuries) with high rates of extrinsic mortality. According to the researchers, <b>extrinsic mortality acts as a confounding factor that masks the genetic signal<\/b>, artificially lowering the impact of genetics.  <\/p>\n<p>Let us imagine we have two identical clocks, built with exactly the same material and the same mechanism (just as identical twins have the same DNA).<\/p>\n<p>If we put both clocks in a protected room, they will stop almost at the same time because of the natural wear of their internal gears. In this case, it is clear that their &#8220;lifespan&#8221; depends on the quality of the parts with which they were built (their genetics). <\/p>\n<p>Now let us imagine putting the same clocks in a crowded square. One is crushed by a car after two days, while the other keeps ticking for ten years until a brick falls and destroys it. If we looked only at the data on when they stopped, we might wrongly conclude that the way they were built (genetics) does not matter at all, since one lasted very little and the other a long time.  <\/p>\n<h2><b>Mathematical models and twin studies<\/b><\/h2>\n<p>Hence the insight. Analyzing the data of three large twin databases (Danish, Swedish and the SATSA study) and of the siblings of US centenarians, the researchers applied <b>Falconer&#8217;s formula<\/b>, which compares the correlation of lifespan between monozygotic (MZ) twins, who share 100% of their genome, and dizygotic (DZ) twins, who share about 50% of it, and used advanced mathematical models to separate the intrinsic and extrinsic components of mortality. <\/p>\n<h2><b>\u201cDNA, half of longevity\u201d<\/b><\/h2>\n<p>Here are the results:<\/p>\n<ol>\n<li aria-level=\"1\"><b>Estimates doubled:<\/b> As anticipated, when extrinsic mortality is removed from the calculations, the heritability of longevity rises from the common 20-25% to about <b>55%<\/b>.<\/li>\n<li aria-level=\"1\"><b>Historical validation:<\/b> In the SATSA study, the researchers observed that measured heritability naturally increases over the decades (from 1900 to 1935), as extrinsic mortality in society decreased thanks to medical and social progress.<\/li>\n<li aria-level=\"1\"><b>The Effect of cutoff age:<\/b> The research also clarifies how the minimum age for inclusion in a study (cutoff age) influences the results. In modern populations with low extrinsic mortality, including early deaths is fundamental to capture the genetic signals of intrinsic mortality. <\/li>\n<\/ol>\n<p>&nbsp;<\/p>\n    <div class=\"container-product-box_art\">\n        <div class=\"product-box_art\">\n            <div class=\"product-image_art\"><img fetchpriority=\"high\" fetchpriority=\"high\" decoding=\"async\" width=\"1920\" height=\"1080\" src=\"https:\/\/solongevity.com\/wp-content\/uploads\/2025\/08\/solongevity-metodo-hero.jpg\" class=\"attachment-full size-full wp-post-image\" alt=\"\" srcset=\"https:\/\/solongevity.com\/wp-content\/uploads\/2025\/08\/solongevity-metodo-hero.jpg 1920w, https:\/\/solongevity.com\/wp-content\/uploads\/2025\/08\/solongevity-metodo-hero-1280x720.jpg 1280w, https:\/\/solongevity.com\/wp-content\/uploads\/2025\/08\/solongevity-metodo-hero-980x551.jpg 980w, https:\/\/solongevity.com\/wp-content\/uploads\/2025\/08\/solongevity-metodo-hero-480x270.jpg 480w\" sizes=\"(min-width: 0px) and (max-width: 480px) 480px, (min-width: 481px) and (max-width: 980px) 980px, (min-width: 981px) and (max-width: 1280px) 1280px, (min-width: 1281px) 1920px, 100vw\" \/><\/div>\n            <div class=\"product-details_art\">\n                <h2 class=\"product-title_art\">Il metodo SoLongevity Clinic<\/h2>\n                <div class=\"product-description_art\">Il metodo SoLongevity \u00e8 messo a punto per agire in modo specifico sugli aspetti che determinano la longevit\u00e0 in salute oltre alla genetica. Per esempio per agire a livello epigenetico e massimizzare il potenziale di longevit\u00e0 scritta nel DNA.<\/div>\n                <a href=\"https:\/\/solongevity.com\/clinica\/metodo-solongevity-clinic\/\" class=\"button product-button\">Vai alla pagina<\/a>\n            <\/div>\n        <\/div>\n    <\/div>\n    \n<h2><b>Why is this research important?<\/b><\/h2>\n<p>These conclusions are surprising because they align the heritability of human longevity with that observed in other species, such as laboratory mice, and with most other human physiological traits.<\/p>\n<p>This study <b>strengthens the rationale behind large-scale efforts to identify genetic variants associated with longevity<\/b> and to link these differences to specific biological pathways that regulate aging. If genetics explains half of the variation in our \u201cbiological\u201d lifespan, identifying the genes involved could revolutionize preventive medicine and public health. <\/p>\n<h2><b>A note of caution<\/b><\/h2>\n<p>Although the genetic component is stronger than thought, <b>50% of the variation remains unexplained<\/b>. This &#8220;missing&#8221; part is attributable to environmental influences (lifestyle, socioeconomic factors), to <b>biological stochasticity<\/b> <b>intrinsic<\/b> to the organism (even if two cells have the same DNA and are in the same environment, they can react slightly differently) and to <b>epigenetic modifications <\/b>(which influence gene expression). Genetics, in short, is not a destiny already written, but a fundamental foundation that we now know to be much more relevant than science had previously assumed.  <\/p>\n<blockquote><p>The discovery makes the interventions that can be implemented today to enhance positive genetic traits and limit negative ones even more important. This is the goal and philosophy of the SoLongevity method and of the SoLongevity Clinic in Milan. <\/p><\/blockquote>\n<h2><b>A study that changes the paradigm<\/b><\/h2>\n<p>\u201cFor those who work in longevity medicine, these results are a radical paradigm shift \u2014 comments <a href=\"https:\/\/solongevity.com\/en\/scientific-committee-2\/alberto-beretta\/\" target=\"_blank\" rel=\"noopener\"><b>Alberto Beretta<\/b><\/a>, President and Scientific Director of <strong>SoLongevity<\/strong> \u2014. While on the one hand it is true and scientifically established that lifestyle and the environment \u2014 the so-called <b>exposome <\/b>\u2014 in which we live are decisive, on the other hand we cannot imagine practicing longevity medicine without periodically updating our knowledge of the genes that determine it. <\/p>\n<p>And here I would like to clarify a fundamental concept. The \u2018<b>genetics of longevity<\/b>\u2019 \u2014 or, if you want to reverse the concept, the \u2018<b>genetics of mortality<\/b>\u2019 \u2014 is the result of the combination of two different categories of genes: those that control the intrinsic mechanisms of biological aging (the famous <b>hallmarks of aging<\/b>) and those that control the way and timing in which human beings develop age-related diseases. <\/p>\n<p>On the former we still have partial knowledge, focused on 3 or 4 apparently dominant genes that are still difficult to interpret in clinical practice. Studies on <b>centenarians <\/b>will guide us in this field. On the latter, instead, we have data available on thousands of genes which, analyzed as a whole, provide us with very useful information for stratifying a person&#8217;s specific risk of developing a certain disease.  <\/p>\n<p>When we manage to integrate these two types of genetic information, we will finally be able to understand why 50% and not 5% of aging is determined by genetics. It seems difficult but, with the arrival of artificial intelligence, the goal is now within our reach\u201d. <\/p>\n","protected":false},"excerpt":{"rendered":"<p>A study conducted on pairs of twins raises the role of genetics in determining lifespan: from less than 25% to over 50%. The comment of Alberto Beretta, President and Scientific Director of SoLongevity <\/p>\n","protected":false},"author":10,"featured_media":0,"comment_status":"closed","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"_et_pb_use_builder":"","_et_pb_old_content":"","_et_gb_content_width":"","footnotes":""},"categories":[158],"tags":[330,332,331,267],"class_list":["post-40731","post","type-post","status-publish","format-standard","hentry","category-news-en","tag-dna","tag-genetics","tag-lifespan","tag-longevity"],"acf":[],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v28.1 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>Is Longevity Written in Our DNA? Here Is How Much Genetics Really Matters in the Length of Our Lives - SoLongevity<\/title>\n<meta name=\"description\" content=\"Is longevity written in DNA? 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