As we age, some cells of the immune system seem to lose their memory and revert to resembling those of infants. This makes them less effective in responding to previously encountered pathogens and could have implications in the development of new vaccines
To function well, the immune system, and particularly its cellular components, need to maintain a proper energy balance
The immune system protects us from attack by pathogens through two types of responses: innate and acquired immunity. The first is very quick and powerful, but not specific. The second, in contrast, is slower and is specific. The latter is based on the activation of highly specialized cells, B lymphocytes and T lymphocytes
Several studies support the hypothesis that the efficacy of immunotherapy can be prolonged over time by acting on mechanisms that regulate intracellular oxidative stress
The immune system protects us from potential threats, but if it is not functioning at its best it can give rise to exaggerated processes of inflammation and oxidation, which damage the body and promote aging and related diseases. “Feeding” it correctly is imperative
Memory and concentration issues, fogginess, anxiety, depression, and loss of taste and smell: these are all neurocognitive symptoms of what is known as Long Covid Syndrome.
According to some clinical observations, the immune system of young Covid-19 survivors appears to age prematurely, contributing to the onset of Long Covid. But this ageing process could also be reversed
Solongevity Research, published by the international scientific journal in Immunology, addresses the issue of interactions between SARS-CoV-2 and other seasonal coronaviruses, immunological interactions that may explain the severe clinical course of some patients and the seasonal pattern.
As the immune system ages, it undergoes alterations and becomes less efficient. This is how processes such as immunosenescence and inflammaging affect it, exposing us to an increased risk of infection by pathogens such as the new coronavirus SARS-CoV-2.
The elderly are more susceptible to infections such as coronavirus SARS-CoV-2 due to phenomena such as immunosenescence and inflammation. At their origin, however, there are epigenetic changes that we can influence through our lifestyle.
